Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2002 1
2004 1
2007 1
2009 1
2011 1
2012 1
2013 1
2014 1
2016 1
2017 1
2020 1
2021 2
2022 2
2023 1
2024 0

Text availability

Article attribute

Article type

Publication date

Search Results

16 results

Results by year

Filters applied: . Clear all
Page 1
A myeloid tumor suppressor role for NOL3.
Stanley RF, Piszczatowski RT, Bartholdy B, Mitchell K, McKimpson WM, Narayanagari S, Walter D, Todorova TI, Hirsch C, Makishima H, Will B, McMahon C, Gritsman K, Maciejewski JP, Kitsis RN, Steidl U. Stanley RF, et al. J Exp Med. 2017 Mar 6;214(3):753-771. doi: 10.1084/jem.20162089. Epub 2017 Feb 23. J Exp Med. 2017. PMID: 28232469 Free PMC article.
Recent discoveries have identified underlying disease-modifying molecular aberrations contributing to disease initiation and progression. Here, we report that deletion of Nol3 (Nucleolar protein 3) in mice leads to an MPN resembling primary myelofibros …
Recent discoveries have identified underlying disease-modifying molecular aberrations contributing to disease initiation and progression. He …
Tat-NOL3 protects against hippocampal neuronal cell death induced by oxidative stress through the regulation of apoptotic pathways.
Sohn EJ, Shin MJ, Eum WS, Kim DW, Yong JI, Ryu EJ, Park JH, Cho SB, Cha HJ, Kim SJ, Yeo HJ, Yeo EJ, Choi YJ, Im SK, Kweon HY, Kim DS, Yu YH, Cho SW, Park M, Park J, Cho YJ, Choi SY. Sohn EJ, et al. Int J Mol Med. 2016 Jul;38(1):225-35. doi: 10.3892/ijmm.2016.2596. Epub 2016 May 19. Int J Mol Med. 2016. PMID: 27221790
Oxidative stress-induced apoptosis is associated with neuronal cell death and ischemia. The NOL3 [nucleolar protein 3 (apoptosis repressor with CARD domain)] protein protects against oxidative stress-induced cell death. However, the protective m …
Oxidative stress-induced apoptosis is associated with neuronal cell death and ischemia. The NOL3 [nucleolar protein
Possible implication of miR-142-3p in coronary microembolization induced myocardial injury via ATXN1L/HDAC3/NOL3 axis.
Xu Y, Lv X, Cai R, Ren Y, He S, Zhang W, Li Q, Yang X, Dai R, Wei R, Su Q. Xu Y, et al. J Mol Med (Berl). 2022 May;100(5):763-780. doi: 10.1007/s00109-022-02198-z. Epub 2022 Apr 12. J Mol Med (Berl). 2022. PMID: 35414011
Immunofluorescence determined the localization of histone deacetylase 3 (HDAC3). The interaction between miR-142-3p and ATXN1L as well as the binding between HDAC3 and histone 3 (H3) was identified. The binding of ATXN1L and HDAC3 to NOL3 promoter was verifie …
Immunofluorescence determined the localization of histone deacetylase 3 (HDAC3). The interaction between miR-142-3p and ATXN1L as wel …
Evaluation of autoantibody signatures in pituitary adenoma patients using human proteome arrays.
Banerjee A, Ray A, Barpanda A, Dash A, Gupta I, Nissa MU, Zhu H, Shah A, Duttagupta SP, Goel A, Srivastava S. Banerjee A, et al. Proteomics Clin Appl. 2022 Nov;16(6):e2100111. doi: 10.1002/prca.202100111. Epub 2022 Sep 18. Proteomics Clin Appl. 2022. PMID: 35939377
RESULTS: Autoantibody profile of PAs led to the identification of differentially expressed significant proteins such as AKNAD1 (AT-Hook Transcription Factor [AKNA] Domain Containing 1), NINJ1 (Nerve injury-induced protein 1), L3HYPDH (Trans-3-hydroxy-L-prolin …
RESULTS: Autoantibody profile of PAs led to the identification of differentially expressed significant proteins such as AKNAD1 (AT-Ho …
MicroRNA expression profiles of circulating microvesicles in hepatocellular carcinoma.
Sun L, Hu J, Xiong W, Chen X, Li H, Jie S. Sun L, et al. Acta Gastroenterol Belg. 2013 Dec;76(4):386-92. Acta Gastroenterol Belg. 2013. PMID: 24592541 Free article.
Among them, 115 miRNAs were over-expressed with up to 31 fold difference (miR-671-5p) and 127 were down-expressed with up to 0.041 fold difference (miR-432) in HCC. By software miRror2.0, nucleolar protein 3 (NOL3) was found to be the core player among …
Among them, 115 miRNAs were over-expressed with up to 31 fold difference (miR-671-5p) and 127 were down-expressed with up to 0.041 fold diff …
Expression of apoptotic tumour necrosis factor receptor-associated factor, caspase recruitment domain and cell death-inducing DFF-45 effector genes in therapy-treated renal cell carcinoma.
Rajandram R, Pat BK, Li J, Johnson DW, Gobe GC. Rajandram R, et al. Nephrology (Carlton). 2009 Apr;14(2):205-12. doi: 10.1111/j.1440-1797.2008.01027.x. Nephrology (Carlton). 2009. PMID: 19076291
These were the tumour necrosis factor receptor-associated factors (TRAF1, 3 and 4), caspase recruitment domain (NOL3 and PYCARD), and cell death-inducing DFF-45 effector domain (ICAD/CAD) genes. ...TRAF1 had significantly increased expression for RNA and protein
These were the tumour necrosis factor receptor-associated factors (TRAF1, 3 and 4), caspase recruitment domain (NOL3 and PYCAR …
Discovery and construction of prognostic model for clear cell renal cell carcinoma based on single-cell and bulk transcriptome analysis.
Zhang F, Yu S, Wu P, Liu L, Wei D, Li S. Zhang F, et al. Transl Androl Urol. 2021 Sep;10(9):3540-3554. doi: 10.21037/tau-21-581. Transl Androl Urol. 2021. PMID: 34733651 Free PMC article.
Besides, GO analysis revealed that biological processes were different between the two subgroups. Egl-9 family hypoxia-inducible factor 3 (EGLN3) and nucleolar protein 3 (NOL3) were specifically expressed in cancer cell clusters, bulk RNA sequen …
Besides, GO analysis revealed that biological processes were different between the two subgroups. Egl-9 family hypoxia-inducible factor 3
Apoptosis repressor with caspase recruitment domain promotes cell proliferation and phenotypic modulation through 14-3-3epsilon/YAP signaling in vascular smooth muscle cells.
Liu M, Yu T, Li M, Fang X, Hou B, Liu G, Wang J. Liu M, et al. J Mol Cell Cardiol. 2020 Oct;147:35-48. doi: 10.1016/j.yjmcc.2020.08.003. Epub 2020 Aug 6. J Mol Cell Cardiol. 2020. PMID: 32771410
Immunofluorescence staining was used to detect the protein location, and immunohistochemistry was used to detect protein expression in tissues. ...Our results also showed that ARC promoted cell proliferation and induced phenotypic modulation of VSMCs in vitro and vi …
Immunofluorescence staining was used to detect the protein location, and immunohistochemistry was used to detect protein expre …
Cell death-resistance of differentiated myotubes is associated with enhanced anti-apoptotic mechanisms compared to myoblasts.
Xiao R, Ferry AL, Dupont-Versteegden EE. Xiao R, et al. Apoptosis. 2011 Mar;16(3):221-34. doi: 10.1007/s10495-010-0566-9. Apoptosis. 2011. PMID: 21161388 Free PMC article.
However, exposure to H(2)O(2) did not lead to caspase-3 activation in myoblasts, and only to a modest induction in myotubes. ...Moreover, increased expression of HSPs in myoblasts attenuated cell death in response to H(2)O(2) and Stsp. Protein abundance of the pro-a …
However, exposure to H(2)O(2) did not lead to caspase-3 activation in myoblasts, and only to a modest induction in myotubes. ...Moreo …
16 results